The Cholesterol Heart Disease Connection There is no clear scientifically proven explanation as to precisely how a diet high in saturated fat could be the cause of rising cholesterol levels in the blood. We also do not know precisely how cholesterol could develop heart disease. But there is a very plausible theory that has a large following in the scientific community. The LDL connection It begins with an increased level of LDL cholesterol. Excess levels of LDL cholesterol usually result in some of the excess cholesterol to move towards the artery walls. The more the LDL levels, the more cholesterol moves out of the bloodstream and into the blood vessel walls. Rigidity of the artery walls may determine how much cholesterol may adhere to the artery walls. Smoking, high blood pressure, diabetic issues, as well as other influences such as stress might constrict artery walls. This may result in a disruption in the flow of blood and pressure. When this happens, artery walls can weaken or become scarred in the first layer of the lining allowing LDL to even further embed into the artery walls. The role of swelling This sets off a sequence of events in which the body effectively sabotages itself. Swelling flares up in the affected region causing White blood cells or macrophages to rush to the scene. The macrophages ingest the cholesterol and become engorged further blocking the arteries. These cells continue to demand even more reinforcements causing far more congestion for the blood circulation. The macrophages are created to destroy infectious microbes and then disappear are fighting Lipids which by their very nature are being continually reproduced. A never ending battle rages. This leads to a steady and continuing state of swelling inside the artery wall. Gradually the overloaded macrophage is destroyed and all the cholesterol and inflammatory substances are discharged into the artery wall. Eventually the body forms a cap of sorts over the inflamed wall section. This cap forms plaque which begins the state of atherosclerosis and the shrinking of the artery opening and resultant constriction in the flow of blood. If this occur in an artery that leads to the heart, it may impede the blood circulation to the heart. This restriction however does not usually cause a cardiac arrest. Plaque deposits are full of inflammation and LDL. Immune cells, T-lymphocytes and macrophages, are the most severe in terms of containing inflammatory cells. If the plaque deposit has a thin cap, it is more likely to rupture. Ruptures are the most common cause of heart attacks. If a plaque cap rupture, blood will seep into the artery wall. The normal wound response mechanism starts and clotting agents are transported to the region. Platelets enter the wound to create a scab that inside an artery wall is life threatening. This scab or clot inside an artery is called a thrombus. It can cut off flow of blood and oxygen to the heart muscle. The section of the heart which is lacking oxygen rich blood starts to die. This is why cardiac arrest occur. The medical term is a myocardial infarction. The Seriousness Of Knowing About Cholesterol Prior To Going On A Cholesterol Controlling Medicine Because too much cholesterol can damage the heart and the rest of the circulatory system, we are strongly urged to keep our cholesterol at the proper levels. When you have changed what you can, if your lipid and cholesterol numbers are still in an unhealthy range, you and your doctor may decide on using a cholesterol drug. A Quick Guide On How You Can Maintain And Lower Bad Cholesterol Bad cholesterol, which is also known as low-density lipoprotein (LDL), is a bad element because the molecules cannot dissolve in the blood and these have a tendency to cling to artery walls potentially provoking a heart attack or causing other heart related diseases. 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